According to them, amyloid from the brain can stimulate the growth of fibrils in the pancreas and pancreatic-related amyloid can be found along with brain-related amyloid in human brain senile plaques.

The research sought to uncover how the two diseases are connected by a process called amyloidosis which occurs in both Alzheimer's and diabetes patients.

The investigation focused on understanding how amyloid deposits 'seed' or spread within a tissue or from one organ to another.

IAPP has binding sites in the brain that are suspected to play a role in satiety and emptying of the stomach. If this is the case, then it might explain where these amyloid deposits come from as well as why Type 2 diabetics are more than twice as likely to develop Alzheimer's.

IAPP may have the ability to travel between the pancreas and the brain, building plaques in both. The study appeared in the American Journal of Pathology.

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